Menu

Publications

2015

  1. Adachi, J.*, Kishida, M., Watanabe, S., Hashimoto, Y., Fukamizu, K., and Tomonaga, T. (2015). Data for Proteomic Analysis of ATP-Binding Proteins and Kinase Inhibitor Target Proteins Using an ATP Probe. Data in Brief, 726-729. DOI: 10.1016/j.dib.2015.10.018
  2. Kazami, T., Nie, H., Satoh, M., Kuga, T., Matsushita, K., Kawasaki, N., Tomonaga, T.*, and Nomura, F. (2015). Nuclear accumulation of annexin A2 contributes to chromosomal instability by coilin-mediated centromere damage. Oncogene 34, 4177-4189. DOI: 10.1038/onc.2014.345
  3. Sato, M.*, Matsubara, T., Adachi, J., Hashimoto, Y., Kishida, M., Yang, Y., Wakefield, L.M., and Tomonaga, T.* (2015) Differential proteome analysis identifies TGF-β-related pro-metastatic proteins in a 4T1 murine breast cancer model. PLoS One 10, e0126483. DOI: 10.1371/journal.pone.0126483
  4. Aoyama, K., Yamaguchi, N., Yuki, R., Morii, M., Kubota, S., Hirata, K., Abe, K., Honda, T., Kuga, T., Hashimoto, Y., and Tomonaga, T. (2015). c-Abl induces stabilization of histone deacetylase 1 (HDAC1) in a kinase activity-dependent manner. Cell Biol Int 39, 446-456. DOI: 10.1002/cbin.10413
  5. Kubota, S., Morii, M., Yuki, R., Yamaguchi, N., Yamaguchi, H., Aoyama, K., Kuga, T., Tomonaga, T., and Yamaguchi, N. (2015). Role for Tyrosine Phosphorylation of A-kinase Anchoring Protein 8 (AKAP8) in its Dissociation from Chromatin and the Nuclear Matrix. J Biol Chem 290, 10891-10904. DOI: 10.1074/jbc.M115.643882
  6. Yamaguchi, N., Yuki, R., Kubota, S., Aoyama, K., Kuga, T., Hashimoto, Y., and Tomonaga, T. (2015). c-Abl-mediated tyrosine phosphorylation of JunB is required for Adriamycin-induced expression of p21. Biochem J 471, 67-77. DOI: 10.1042/BJ20150372
  7. Takeuchi, M., Higashino, A., Takeuchi, K., Hori, Y., Koshiba-Takeuchi, K., Makino, H., Monobe, Y., Kishida, M., Adachi, J., Takeuchi, J., Tomonaga, T., Umezawa, A., Kameoka, Y., and Akagi, K. (2015). Transcriptional Dynamics of Immortalized Human Mesenchymal Stem Cells during Transformation. PloS one 10, e0126562. DOI: 10.1371/journal.pone.0126562
  8. Matsushita, K., Kitamura, K., Rahmutulla, B., Tanaka, N., Ishige, T., Satoh, M., Hoshino, T., Miyagi, S., Mori, T., Itoga, S., Shimada, H., Tomonaga, T., Kito, M., Nakajima-Takagi, Y., Kubo, S., Nakaseko, C., Hatano, M., Miki, T., Matsuo, M., Fukuyo, M., Kaneda, A., Iwama, A., and Nomura, F. (2015). Haploinsufficiency of the c-myc transcriptional repressor FIR, as a dominant negative-alternative splicing model, promoted p53-dependent T-cell acute lymphoblastic leukemia progression by activating Notch1. Oncotarget 6, 5102-5117. DOI: 10.18632/oncotarget.3244